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Артериальная гипотония у больных с вторичным гиперальдостеронизмом: синдромы Барттера и Гительмана
Артериальная гипотония у больных с вторичным гиперальдостеронизмом: синдромы Барттера и Гительмана
Материалы доступны только для специалистов сферы здравоохранения. Авторизуйтесь или зарегистрируйтесь.
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Список литературы
1. Bartter FC, Pronove P, Gill JR et al. Hyperplasia of the juxtaglomerular complex with hyperaldosteronism and hypokcalemic alkalosis. A new syndrome. Am J Med 1962; 33: 811–28.
2. Gitelman HJ, Graham JB, Welt LG. A new familial disorder Characterized by hypokcalemia and hypomagnesemia. Trans Assoc Am Physicians 1966; 79: 221–35.
3. Simon DB, Nelson-Williams C, Bia MJ et al. Gitleman's variant of Bartter's syndrome, inherited hypokcalemic alkalosis, in caused by mutations in the thiazide-sensitive Na-Cl cotransporter. Nat Genet 1996; 12: 24–30.
4. Simon DB et al. Bartter's syndrome, hypokcalemic alkalosis with hypercaiciuria is caused by mutations in the Na-K-2Cl cotransporter NKCC2. Nat Genet 1996; 13: 183–8.
4. Calo LA. Vascular tone control in humans: insights from studies in Bartter's/Gitelman's syndromes. Kidney Int 2006; 69: 963–6.
5. Bhandari S, Turney JH. The molecular basis of hypocaliemic alkaloses: Bartter's and Gitelman's syndromes. Nephron 1998; 80: 373–9.
6. Pachulski RT, Lopez F, Shazaf R. Gitelman's not-so-benign syndrome. New Eng J Med 2005; 335 (8): 850–1.
7. Sartori M, Parotto E, Bonso E et al. Autonomic nervous system function in chronic hypotension associated with Bartter and Gitelman syndromes. Am J Kidney Dis 2007; 49 (2): 330–5.
8. Calo LA, Davis PA, Milani M et al. Increased endothelial nitric oxide synthase mRNA level in Bartter's and Citelman's syndrome. Relationship to vascular reactivity. Clin Nephrol 1999; 51: 12–7.
2. Gitelman HJ, Graham JB, Welt LG. A new familial disorder Characterized by hypokcalemia and hypomagnesemia. Trans Assoc Am Physicians 1966; 79: 221–35.
3. Simon DB, Nelson-Williams C, Bia MJ et al. Gitleman's variant of Bartter's syndrome, inherited hypokcalemic alkalosis, in caused by mutations in the thiazide-sensitive Na-Cl cotransporter. Nat Genet 1996; 12: 24–30.
4. Simon DB et al. Bartter's syndrome, hypokcalemic alkalosis with hypercaiciuria is caused by mutations in the Na-K-2Cl cotransporter NKCC2. Nat Genet 1996; 13: 183–8.
4. Calo LA. Vascular tone control in humans: insights from studies in Bartter's/Gitelman's syndromes. Kidney Int 2006; 69: 963–6.
5. Bhandari S, Turney JH. The molecular basis of hypocaliemic alkaloses: Bartter's and Gitelman's syndromes. Nephron 1998; 80: 373–9.
6. Pachulski RT, Lopez F, Shazaf R. Gitelman's not-so-benign syndrome. New Eng J Med 2005; 335 (8): 850–1.
7. Sartori M, Parotto E, Bonso E et al. Autonomic nervous system function in chronic hypotension associated with Bartter and Gitelman syndromes. Am J Kidney Dis 2007; 49 (2): 330–5.
8. Calo LA, Davis PA, Milani M et al. Increased endothelial nitric oxide synthase mRNA level in Bartter's and Citelman's syndrome. Relationship to vascular reactivity. Clin Nephrol 1999; 51: 12–7.
Авторы
Н.М.Чихладзе
Отдел системных гипертензий ФГУ РКНПК Росмедтехнологий, Москва
Отдел системных гипертензий ФГУ РКНПК Росмедтехнологий, Москва
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